Preview

Acta Biomedica Scientifica

Advanced search

GRADED CRITERIA OF DIAGNOSTICS AND PREDICTION OF CENTRAL SEROUS CHORIORETINOPATHY OUTCOME

https://doi.org/10.12737/23794

Abstract

Present concept of central serous chorioretinopathy classification does not reflect the true picture of the pathological process. The purpose of the study was the development of diagnostic criteria of central serous chorioretinopathy clinical forms and basing on that extend and optimize its classification with assessment of risk factors and prognosis of disease. Full ophthalmologic and somatic examination of 56 men with central serous chorioretinopathy was made. Focal laser coagulation was performed in patients with fluorescein leakage and local injury of pigment epithelium by angiographic data (22 patients). In the case of extended damage of pigment epithelium and diffuse leakage of fluorescein (23 people) transpupillary thermotherapy of optical disk was conducted. Detailed expert evaluation of retinal changes by fluorescein angiographic data allowed dividing the patients into 3 groups. The main criterion for the division was the damaged area of the pigment epithelium: group 1 - central serous chorioretinopathy with local injury of pigment epithelium (with leakage point); group 2 - central serous chorioretinopathy with extensive damage of pigment epithelium with diffuse leakage; group 3 - central serous chorioretinopathy with extensive damage of pigment epithelium combined with leakage point (recurrence of leakage). The results of our study brought us to the following conclusion. Development of new classification criteria of the disease allows to construct a more specific algorithm of therapeutic measures and to determine prognosis of central serous chorioretinopathy already at the stage of diagnostics.

About the Authors

A. G. Shchuko
Иркутский филиал ФГАУ «МНТК «Микрохирургия глаза» им. акад. С.Н. Федорова» МЗ РФ; ГБОУ ДПО «Иркутская государственная медицинская академия последипломного образования» Минздрава России
Russian Federation


A. N. Zlobina
Иркутский филиал ФГАУ «МНТК «Микрохирургия глаза» им. акад. С.Н. Федорова» МЗ РФ
Russian Federation


V. V. Bukina
Иркутский филиал ФГАУ «МНТК «Микрохирургия глаза» им. акад. С.Н. Федорова» МЗ РФ
Russian Federation


A. A. Shchuko
Иркутский филиал ФГАУ «МНТК «Микрохирургия глаза» им. акад. С.Н. Федорова» МЗ РФ
Russian Federation


References

1. Волкова Н.В., Щуко А.Г., Малышев В.В. Ретроспективный анализ факторов риска развития рубцовых изменений путей оттока внутриглазной жидкости после фистулизирующих антиглаукоматозных операций. Сообщение 1 // Национальный журнал Глаукома. - 2010. - № 3. - С. 35-40. Volkova NV, Shchuko AG, Malyshev VV (2010). Retrospective analysis of risk factors for the development of scar changes the intraocular fluid outflow pathways after fistulizing antiglaucomatous surgery. Report 1 [Retrospektivnyy analiz faktorov riska razvitiya rubtsovykh izmeneniy putey ottoka vnutriglaznoy zhidkosti posle fistuliziruyushchikh antiglaukomatoznykh operatsiy. Soobshchenie 1]. Natsional'nyy zhurnal Glaukoma, (3), 35-40.

2. Волкова Н.В., Щуко А.Г., Носкова Л.К., Малышев В.В. Роль гиперлипопероксидации и исходной гормональной регуляции в механизмах рубцовых изменений путей оттока внутриглазной жидкости после фистулизирующих антиглаукоматозных операций. Сообщение 2 // Национальный журнал Глаукома. -2011. - Т. 10, № 2. - С. 3-7. Volkova NV, Shchuko AG, Noskova LK, Malyshev VV (2011). Role of hyperlipoperoxidation and initial hormonal regulation in the mechanisms of scarring changes of intraocular fluid outflow pathways after fistulizing antiglaucomatous surgery. Report 2 [Rol’ giperlipoperoksidatsii i iskhodnoy gormonal’noy regulyatsii v mekhanizmakh rubtsovykh izmeneniy putey ottoka vnutriglaznoy zhidkosti posle fistuliziruyushchikh antiglaukomatoznykh operatsiy. Soobshchenie 2]. Natsional'nyy zhurnal Glaukoma, 10 (2). 3-7.

3. Кацнельсон Л.А., Форофонова Т.И., Бунин А.Я. Сосудистые заболевания глаз. - М.: Медицина, 1990. -193 c. Katznelson LA, Forofonova TI, Bunin AY (1990). Vascular diseases of the eyes [Sosudistye zabolevaniya glaz], 193.

4. Gass JD (1991). Central serous chorioretinopathy and white subretinal exudation during pregnancy. Arch. Ophthalmol, (109), 677-681.

5. Guyer DR, Yannuzzi LA, Slakter JS, Sorenson JA, Ho A, Orlock D (1994). Digital indocyanine green videoangiography of central serous chorioretinopathy. Arch. Ophthalmol., (112), 1057-1062.

6. Iida T, Kishi S, Hagimura N, Shimizu K (1999). Persistent and bilateral choroidal vascular abnormalities in central serous chorioretinopathy. Retina, (19), 508-512.

7. Imamura Y, Fujiwara T, Margolis R, Spaide RF (2009). Enhanced depth imaging optical coherence tomography of the choroid in central serous chorioretinopathy. Retina, (29), 1469-1473.

8. Marmor MF (1988). New hypothesis on the pathogenesis and treatment of serous retinal detachment. Graefes Arch. Clin. Exp. Ophthalmol., (226), 548-552.

9. Moschos M, Brouzas D, Koutsandrea C, Stefanos B, Loukianou H, Papantonis F, Moschos M (2007). Assessment of central serous chorioretinopathy by optical coherence tomography and multifocal electroretinography. Ophthalmologica, 221 (5), 292-298.

10. Sekiryu T, Iida T, Maruko I, Saito K, Kondo T (2010). Infrared fundus autofluorescence and central serous chorioretinopathy. Investigative Ophthalmology & Visual Science, 51 (10), 4956-4962.

11. Yannuzzi LA (1986). Type A behavior and central serous chorioretinopathy. Trans. Am. Ophthalmol. Soc., (84), 799-845.


Review

For citations:


Shchuko A.G., Zlobina A.N., Bukina V.V., Shchuko A.A. GRADED CRITERIA OF DIAGNOSTICS AND PREDICTION OF CENTRAL SEROUS CHORIORETINOPATHY OUTCOME. Acta Biomedica Scientifica. 2016;1(6):131-136. (In Russ.) https://doi.org/10.12737/23794

Views: 617


Creative Commons License
This work is licensed under a Creative Commons Attribution 4.0 License.


ISSN 2541-9420 (Print)
ISSN 2587-9596 (Online)